Motor Speech Disorders (Dysarthria Types & CAS)
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Dysarthria vs. Apraxia of Speech
| Feature | Dysarthria | Apraxia of Speech (AOS) |
|---|---|---|
| Nature | Neuromuscular execution disorder | Motor planning/programming disorder |
| Errors | Consistent; distortions predominate | Distortions and distorted substitutions; consistency is debated (ASHA lists inconsistency; other criteria describe errors as relatively consistent in type and location) |
| Rate | Often slow or rapid (type-dependent) | Slow with trial-and-error groping |
| Prosody | Abnormal (type-specific) | Disrupted; equal stress pattern |
| Automatic speech | Equally affected | Better than volitional speech |
| Error pattern | Imprecise production from reduced strength, speed, range, or coordination | Errors increase with word length/complexity |
| Nonspeech oral movements | Often impaired | May be intact (dissociation) |
High-Yield: AOS is a PLANNING disorder, not a muscle weakness disorder. Hallmarks: slow rate with lengthened sounds and pauses, sound distortions and distorted substitutions, syllable segmentation with equalized stress, groping, and more errors as length and complexity rise; automatic speech is often easier. Error consistency is debated: ASHA lists inconsistent errors, while other criteria describe errors as relatively consistent in type and location.
Dysarthria Types (Mayo Classification)
| Type | Lesion Site | Key Features | Associated Conditions |
|---|---|---|---|
| Flaccid | LMN (cranial/spinal nerves) | Hypernasality, breathy voice, nasal emission, muscle atrophy, fasciculations | Myasthenia gravis, Guillain-Barré, Bell's palsy, progressive bulbar palsy |
| Spastic | UMN (bilateral) | Strained-strangled voice, slow rate, hypernasality, reduced range | Bilateral stroke, pseudobulbar palsy, spastic CP |
| Ataxic | Cerebellum | Irregular articulatory breakdowns, excess and equal stress, scanning speech, "drunken" quality | Cerebellar stroke, MS, Friedreich's ataxia, alcohol toxicity |
| Hypokinetic | Basal ganglia (substantia nigra) | Monopitch, monoloudness, reduced stress, short rushes of speech, breathy voice, festinating speech | Parkinson's disease |
| Hyperkinetic | Basal ganglia | Involuntary movements affect speech; variable; voice stoppages; excess loudness variations | Huntington's disease, dystonia, chorea, Tourette's |
| Unilateral UMN | UMN (unilateral) | Imprecise consonants, harsh voice, slow rate; generally mild | Unilateral stroke |
| Mixed | Multiple systems | Combined features | ALS (flaccid + spastic), MS, Wilson's disease |
High-Yield: ALS = mixed flaccid-spastic dysarthria. Parkinson's = hypokinetic dysarthria. Ataxic dysarthria features "scanning speech" with excess and equal stress.
Childhood Apraxia of Speech (CAS)
ASHA's 3 Consensus Features:
- Inconsistent errors on consonants and vowels in repeated productions
- Lengthened and disrupted coarticulatory transitions between sounds/syllables
- Inappropriate prosody (especially equal stress or lexical stress errors)
Additional CAS Characteristics:
- Limited consonant and vowel inventory
- Vowel errors (unusual in pure articulation disorders)
- Groping/silent posturing
- Increased errors with length and complexity
- Gap between receptive and expressive language (receptive >> expressive)
- Difficulty with volitional oral movements (may co-occur with oral apraxia)
- Repetition of single syllables (/papapa/) may be near normal while sequenced syllables (/pataka/) break down; this AMR/SMR contrast points to planning rather than weakness
CAS Treatment:
- DTTC (Dynamic Temporal and Tactile Cueing): Integral stimulation ("watch me, listen to me, do what I do")
- ReST (Rapid Syllable Transition Treatment): Targets smooth transitions
- PROMPT (Prompts for Restructuring Oral Muscular Phonetic Targets): Tactile-kinesthetic cues
- Frequent, high-repetition practice, with the session schedule set from the child's response and checked with data
- Principles of motor learning: practice amount and distribution, practice variability, and feedback type and frequency, each adjusted to the child
- Treatment approaches, target selection, and dosage for CAS are covered in Speech Sound Disorders: Intervention
High-Yield: CAS is diagnosed by behavioral speech features, NOT by neurological testing. Do NOT diagnose CAS based on non-speech oral motor difficulties alone.
Treatment for Dysarthria
- Lee Silverman Voice Treatment (LSVT LOUD): Hypokinetic dysarthria (Parkinson's); "Think loud!"
- Palatal lift/prosthesis: Flaccid dysarthria with velopharyngeal incompetence
- Rate control strategies: Pacing board, alphabet board, finger tapping
- Respiratory support: Expiratory muscle strength training (EMST), postural adjustments
- Prosodic training: Contrastive stress drills for monotone speech
Sources to Know
- ASHA Practice Portal: Dysarthria in Adults
- ASHA Practice Portal: Acquired Apraxia of Speech
- ASHA Practice Portal: Childhood Apraxia of Speech
- [ASHA (2007). Childhood Apraxia of Speech [Technical Report]](https://www.asha.org/policy/tr2007-00278/)
- Darley, Aronson & Brown (1969). Differential diagnostic patterns of dysarthria. JSHR
- Maas et al. (2008). Principles of motor learning in treatment of motor speech disorders. AJSLP
- Strand (2020). Dynamic Temporal and Tactile Cueing: a treatment strategy for childhood apraxia of speech. AJSLP
- Murray, McCabe & Ballard (2015). A randomized controlled trial comparing Rapid Syllable Transition Treatment and the Nuffield Dyspraxia Programme. JSLHR
- Ramig et al. (2018). Speech treatment in Parkinson's disease: randomized controlled trial. Mov Disord
- Duffy, J. R. (2020). Motor Speech Disorders: Substrates, Differential Diagnosis, and Management (4th ed.). Elsevier.
Reviewed: 2026-09
Key terms 7
- Apraxia of Speech (AOS)
- Motor speech planning/programming disorder with slow rate, distortions and distorted substitutions, syllable segmentation, groping, and prosodic disruption; error consistency is debated; NOT due to muscle weakness
- Flaccid dysarthria
- LMN damage causing hypotonia, breathy voice, hypernasality, nasal emission; seen in myasthenia gravis, bulbar palsy
- Spastic dysarthria
- Bilateral UMN damage causing hypertonia, strained-strangled voice, slow rate; seen in pseudobulbar palsy
- Ataxic dysarthria
- Cerebellar damage causing irregular articulatory breakdowns, excess and equal stress, "scanning speech"
- Hypokinetic dysarthria
- Basal ganglia damage (substantia nigra) causing monopitch, monoloudness, festinating speech; hallmark of Parkinson's disease
- CAS
- Childhood Apraxia of Speech: motor planning disorder with inconsistent errors, disrupted coarticulatory transitions, and inappropriate prosody
- DTTC
- Dynamic Temporal and Tactile Cueing: integral stimulation approach for CAS using multimodal cues and systematic cue reduction
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