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Cat. III · Treatment Planning & Implementation4 min read
Speech Sound Production

Motor Speech Disorders (Dysarthria Types & CAS)

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  1. Dysarthria vs. Apraxia of Speech
  2. Dysarthria Types (Mayo Classification)
  3. Childhood Apraxia of Speech (CAS)
  4. Treatment for Dysarthria
  5. Sources to Know

Dysarthria vs. Apraxia of Speech

FeatureDysarthriaApraxia of Speech (AOS)
NatureNeuromuscular execution disorderMotor planning/programming disorder
ErrorsConsistent; distortions predominateDistortions and distorted substitutions; consistency is debated (ASHA lists inconsistency; other criteria describe errors as relatively consistent in type and location)
RateOften slow or rapid (type-dependent)Slow with trial-and-error groping
ProsodyAbnormal (type-specific)Disrupted; equal stress pattern
Automatic speechEqually affectedBetter than volitional speech
Error patternImprecise production from reduced strength, speed, range, or coordinationErrors increase with word length/complexity
Nonspeech oral movementsOften impairedMay be intact (dissociation)

High-Yield: AOS is a PLANNING disorder, not a muscle weakness disorder. Hallmarks: slow rate with lengthened sounds and pauses, sound distortions and distorted substitutions, syllable segmentation with equalized stress, groping, and more errors as length and complexity rise; automatic speech is often easier. Error consistency is debated: ASHA lists inconsistent errors, while other criteria describe errors as relatively consistent in type and location.

Dysarthria Types (Mayo Classification)

TypeLesion SiteKey FeaturesAssociated Conditions
FlaccidLMN (cranial/spinal nerves)Hypernasality, breathy voice, nasal emission, muscle atrophy, fasciculationsMyasthenia gravis, Guillain-Barré, Bell's palsy, progressive bulbar palsy
SpasticUMN (bilateral)Strained-strangled voice, slow rate, hypernasality, reduced rangeBilateral stroke, pseudobulbar palsy, spastic CP
AtaxicCerebellumIrregular articulatory breakdowns, excess and equal stress, scanning speech, "drunken" qualityCerebellar stroke, MS, Friedreich's ataxia, alcohol toxicity
HypokineticBasal ganglia (substantia nigra)Monopitch, monoloudness, reduced stress, short rushes of speech, breathy voice, festinating speechParkinson's disease
HyperkineticBasal gangliaInvoluntary movements affect speech; variable; voice stoppages; excess loudness variationsHuntington's disease, dystonia, chorea, Tourette's
Unilateral UMNUMN (unilateral)Imprecise consonants, harsh voice, slow rate; generally mildUnilateral stroke
MixedMultiple systemsCombined featuresALS (flaccid + spastic), MS, Wilson's disease

High-Yield: ALS = mixed flaccid-spastic dysarthria. Parkinson's = hypokinetic dysarthria. Ataxic dysarthria features "scanning speech" with excess and equal stress.

Childhood Apraxia of Speech (CAS)

ASHA's 3 Consensus Features:

  1. Inconsistent errors on consonants and vowels in repeated productions
  2. Lengthened and disrupted coarticulatory transitions between sounds/syllables
  3. Inappropriate prosody (especially equal stress or lexical stress errors)

Additional CAS Characteristics:

  • Limited consonant and vowel inventory
  • Vowel errors (unusual in pure articulation disorders)
  • Groping/silent posturing
  • Increased errors with length and complexity
  • Gap between receptive and expressive language (receptive >> expressive)
  • Difficulty with volitional oral movements (may co-occur with oral apraxia)
  • Repetition of single syllables (/papapa/) may be near normal while sequenced syllables (/pataka/) break down; this AMR/SMR contrast points to planning rather than weakness

CAS Treatment:

  • DTTC (Dynamic Temporal and Tactile Cueing): Integral stimulation ("watch me, listen to me, do what I do")
  • ReST (Rapid Syllable Transition Treatment): Targets smooth transitions
  • PROMPT (Prompts for Restructuring Oral Muscular Phonetic Targets): Tactile-kinesthetic cues
  • Frequent, high-repetition practice, with the session schedule set from the child's response and checked with data
  • Principles of motor learning: practice amount and distribution, practice variability, and feedback type and frequency, each adjusted to the child
  • Treatment approaches, target selection, and dosage for CAS are covered in Speech Sound Disorders: Intervention

High-Yield: CAS is diagnosed by behavioral speech features, NOT by neurological testing. Do NOT diagnose CAS based on non-speech oral motor difficulties alone.

Treatment for Dysarthria

  • Lee Silverman Voice Treatment (LSVT LOUD): Hypokinetic dysarthria (Parkinson's); "Think loud!"
  • Palatal lift/prosthesis: Flaccid dysarthria with velopharyngeal incompetence
  • Rate control strategies: Pacing board, alphabet board, finger tapping
  • Respiratory support: Expiratory muscle strength training (EMST), postural adjustments
  • Prosodic training: Contrastive stress drills for monotone speech

Sources to Know

Reviewed: 2026-09

Key terms 7

Apraxia of Speech (AOS)
Motor speech planning/programming disorder with slow rate, distortions and distorted substitutions, syllable segmentation, groping, and prosodic disruption; error consistency is debated; NOT due to muscle weakness
Flaccid dysarthria
LMN damage causing hypotonia, breathy voice, hypernasality, nasal emission; seen in myasthenia gravis, bulbar palsy
Spastic dysarthria
Bilateral UMN damage causing hypertonia, strained-strangled voice, slow rate; seen in pseudobulbar palsy
Ataxic dysarthria
Cerebellar damage causing irregular articulatory breakdowns, excess and equal stress, "scanning speech"
Hypokinetic dysarthria
Basal ganglia damage (substantia nigra) causing monopitch, monoloudness, festinating speech; hallmark of Parkinson's disease
CAS
Childhood Apraxia of Speech: motor planning disorder with inconsistent errors, disrupted coarticulatory transitions, and inappropriate prosody
DTTC
Dynamic Temporal and Tactile Cueing: integral stimulation approach for CAS using multimodal cues and systematic cue reduction

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